pseudomembranous colitis การใช้

• Mild cases of pseudomembranous colitis usually respond to discontinuation of the drug alone.
• After the diagnosis of pseudomembranous colitis has been established, therapeutic measures should be initiated.
• For example, on the pseudomembranous colitis talk page, you might have the following.
• Toxic megacolon is usually a Clostridium difficile " infections, which have led to pseudomembranous colitis.
• Pseudomembranous colitis is a potentially lethal condition commonly associated with clindamycin, but which occurs with other antibiotics, as well.
• Lincosamide antibiotics are one of the classes of antibiotics most associated with pseudomembranous colitis caused by " Clostridium difficile ".
• Metronidazole ( Flagyl ), which is used to treat certain parasitic infections as well as pseudomembranous colitis, causes similar effects to disulfiram.
• Pseudomembranous colitis has been reported with nearly all antibacterial agents, including erythromycin, and may range in severity from mild to life threatening.
• TcdA can induce the physiological changes that occur in " C . difficile " related pseudomembranous colitis ( PMC ), a severe ulceration of the colon.
• Pseudomembranous colitis ( caused by " Clostridium difficile " ) has also developed in some patients; this condition may be fatal in a small number of cases.
• Oral teicoplanin has been demonstrated to be effective in the treatment of pseudomembranous colitis and " Clostridium difficile "-associated diarrhoea, with comparable efficacy with vancomycin.
• The only approved indication for oral vancomycin therapy is in the treatment of pseudomembranous colitis, where it must be given orally to reach the site of infection in the colon.
• Hence, this leads to tumor necrosis factor ? ( TNF ? ) and proinflammatory interleukins being established as the major causative agents of pseudomembranous colitis ( PMC ) and antibiotic-associated diarrhea ( AAD ).
• Pseudomembranous colitis first was described as a complication of " C . difficile " infection in 1978, when a toxin was isolated from patients suffering from pseudomembranous colitis and Koch's postulates were met.
• Pseudomembranous colitis first was described as a complication of " C . difficile " infection in 1978, when a toxin was isolated from patients suffering from pseudomembranous colitis and Koch's postulates were met.
• The first description of FMT was published in 1958 by Ben Eiseman and colleagues, a team of surgeons from Colorado, who treated four critically ill patients with fulminant pseudomembranous colitis ( before " C . difficile " was the known cause ) using fecal enemas, which resulted in a rapid return to health.
• The condition is usually caused by Gram-positive enteric commensal bacteria of the Clostridium difficile " is a species of Gram-positive bacteria that commonly causes severe diarrhea and other intestinal diseases when competing bacteria are wiped out by antibiotics, causing pseudomembranous colitis, whereas Clostridium septicum is responsible for most cases of neutropenic enterocolitis.
• Pseudomembranous colitis can follow, creating generalized inflammation of the colon and the development of " pseudomembrane ", a viscous collection of inflammatory cells, fibrin, and necrotic cells . [ 4 ] Clindamycin-resistant " C . difficile " was reported as the causative agent of large outbreaks of diarrheal disease in hospitals in New York, Arizona, Florida and Massachusetts between 1989 and 1992.
• Like nearly all antibiotics, linezolid has been associated with " Clostridium difficile "-associated diarrhea ( CDAD ) and pseudomembranous colitis, although the latter is uncommon, occurring in about one in two thousand patients in clinical trials . " C . difficile " appears to be susceptible to linezolid " in vitro ", and linezolid was even considered as a possible treatment for CDAD.